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Both these actions result in raised levels of monoamines at the synapse. In addition, drugs that reduce the availability of monoamines cause symptoms of depression. From such observations the monoamine theory of depression and mania was formed: Depression is due to an absolute or relative decrease in monoamines, or a decrease in receptor sensitivity, at certain receptor sites in the brain.
Mania is due to an absolute or relative excess of monoamines or an increase in receptor sensitivity at these sites. Even after years of research, it is still not clear which receptor sites or which monoamines are involved, although noradrenaline, serotonin and dopamine are all implicated.
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Cardiac output is the volume of blood pumped out of the left ventricle every minute and is a function of the stroke volume and heart rate. Stroke volume is the amount of blood ejected by the left ventricle during one contraction and the heart rate is the number of beats per minute.
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There are many drugs in each group.
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Despite evidence for and against, there remains one major problem with the monoamine theory of depression.
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The pain pathways also go to other parts of the brain, such as the hypothalamus, the amygdala and reticular activating system.
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Many non-steroidal anti-inflammatory drugs interact with lithium to increase its plasma concentration and risk of toxicity.
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High concentrations of magnesium in the body can interfere with the function of calcium ions in the heart, skeletal muscle and the central nervous system.
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Class III calcium channel blockers are intermediate in action affecting calcium ion channels in both cardiac muscle cells and smooth muscle cells in blood vessels.
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None of them is as potent as halothane, but they are less likely to cause liver toxicity.
) Use of iodine contrast agents is contraindicated in pregnancy because they can cross the placenta and interfere with foetal thyroid development. Special precautions should be taken in patients with known hypersensitivity to iodine and in those with other allergies, for example to foods or other drugs.
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Adverse effects of flucytosine are gastrointestinal upset, anaemia, neutropenia, thrombocytopenia and alopecia, all mild and reversible. This group of drugs inhibits an enzyme necessary for ergosterol synthesis. Some of these drugs are used orally to treat systemic fungal infections; others are used topically to treat skin and nail infection. Adverse effects of these drugs taken orally are generally mild such as gastrointestinal upset, headache and pruritis.
In some patients, particularly those with HIV and on multiple therapy, more serious exfoliative dermatitis and StevensJohnson syndrome has occurred with fluconazole.
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Pneumocystis pneumonia can be treated with co-trimoxazole, which is a mixture of trimethoprim and sulfamethoxazide.